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Azerbaijan Medical Journal

Aim and Scope

Azerbaijan Medical Journal

The Azerbaijan Medical Journal (AMJ) is a peer-reviewed, open-access monthly publication dedicated to advancing medical science and clinical practice through the dissemination of high-quality, original research. The Journal aims to serve as a global platform for researchers, clinicians, and healthcare professionals to share innovative findings, critical reviews, and evidence-based insights that contribute to the understanding, prevention, diagnosis, and treatment of diseases.

AMJ welcomes submissions across a broad spectrum of medical disciplines, including but not limited to:

The Journal particularly encourages contributions from early-career researchers, multidisciplinary teams, and authors from underrepresented regions. All manuscripts undergo rigorous double-blind peer review to ensure scientific integrity, methodological soundness, and relevance to contemporary medical challenges.We invite authors worldwide to submit original articles, review papers, case reports, brief communications, and study protocols that align with the Journal’s mission of promoting excellence, equity, and accessibility in medical scholarship.

📝 Official Submission Process

Manuscripts must be submitted officially through our online system. Please complete the REGISTRATION FORM on our website first to access your Author Dashboard. You may then submit your manuscript for an initial review by our dedicated editorial team to assess its compliance with our journal's policies and scope.

Step-by-Step Submission Process:
  1. 1. Complete registration Form
  2. 2. Access Your Applicant Dashboard
  3. 3. Submit a new article
  4. 4. Fill Out the Submission Form.
    Please ensure you provide a title and abstract for your article, and upload the required document files.
    Accepted formats are PDF, TXT, and DOCX (up to 10MB per file). You may upload up to 5 files.
    The first document file is mandatory.
💡 Need Help?

For technical assistance with submission or registration, please contact:

For general editorial questions or policy inquiries:

Latest Articles

Azerbaijan Medical Journal

AMJ-11-12-2024-12306
Circadian Disruption of the CLOCK-BMAL1 Axis in Hepatic Stellate Cells Promotes Fibrosis Progression in Non-Alcoholic Steatohepatitis

Author(s): Dr. Helena Costa, Dr. James Wilson

Abstract: Non-alcoholic steatohepatitis (NASH) progression exhibits diurnal patterns, yet the molecular clock's role in hepatic fibrogenesis is unknown. We demonstrate that hepatic stellate cells (HSCs) possess autonomous circadian clocks that regulate collagen synthesis and autophagy. Disruption of Clock or Bmal1 in HSCs through genetic knockout or environmental circadian disruption (chronic jet-lag) accelerated fibrosis in methionine-choline deficient (MCD) diet fed mice. Mechanistically, CLOCK-B...
Read More Azerbaijan Medical Journal • 11-12-2024
AMJ-11-12-2024-12305
Implantable Wireless Neuroprosthesis for Bladder Control After Spinal Cord Injury: A Brain-Bladder Closed-Loop Interface

Author(s): Dr. Marcus Johnson, Dr. Yuki Tanaka

Abstract: Neurogenic bladder dysfunction after spinal cord injury (SCI) severely impacts quality of life. We developed an implantable, wireless neuroprosthesis establishing a closed-loop interface between cortical bladder sensation areas and sacral root stimulators. Using electrocorticography (ECoG) decoding of bladder fullness states and real-time pudendal nerve feedback, the system enabled on-demand, physiological voiding in 6 SCI patients during a 12-month trial. Participants achieved 85% volun...
Read More Azerbaijan Medical Journal • 11-12-2024
AMJ-06-11-2024-12304
Long Non-Coding RNA MALAT1 Inhibition via Antisense Oligonucleotides Attenuates Diabetic Kidney Disease Progression Through Regulation of Endothelial-Mesenchymal Transition

Author(s): Dr. Priya Sharma, Dr. Omar Hassan

Abstract: Endothelial-mesenchymal transition (EndMT) contributes to renal fibrosis in diabetic kidney disease (DKD). We identified the long non-coding RNA MALAT1 as a key regulator of EndMT in high glucose-stimulated human glomerular endothelial cells. MALAT1 sponged miR-145, thereby derepressing ZEB2 and promoting endothelial dysfunction. Systemic administration of gapmer antisense oligonucleotides (ASOs) targeting MALAT1 in db/db diabetic mice reduced albuminuria by 50%, preserved glomerular end...
Read More Azerbaijan Medical Journal • 06-11-2024
AMJ-03-10-2024-12303
Cold Atmospheric Plasma Therapy Induces Immunogenic Cell Death in Chemoresistant Triple-Negative Breast Cancer

Author(s): Dr. Anna Kowalski, Dr. Robert Chen

Abstract: Triple-negative breast cancer (TNBC) lacks targeted therapies and frequently develops chemotherapy resistance. Cold atmospheric plasma (CAP) generates reactive oxygen/nitrogen species (RONS) that selectively kill cancer cells. We demonstrate that CAP treatment of cisplatin-resistant TNBC cell lines and patient-derived xenografts induces immunogenic cell death (ICD), characterized by calreticulin exposure, ATP secretion, and HMGB1 release. CAP generated RONS activated the cGAS-STING pathwa...
Read More Azerbaijan Medical Journal • 03-10-2024
AMJ-03-10-2024-12302
Bioprinted Vascularized Cardiac Patches Using Patient-Specific Induced Pluripotent Stem Cells for Pediatric Congenital Heart Defect Repair

Author(s): Dr. Emily Roberts

Abstract: Surgical repair of congenital heart defects (CHD) in neonates is limited by donor tissue availability. We bioprinted vascularized cardiac patches using patient-specific iPSC-derived cardiomyocytes, fibroblasts, and endothelial cells embedded in a decellularized myocardial matrix. Sacrificial Pluronic F127 channels enabled spontaneous microvascular network formation perfusable within 7 days. Patches exhibited synchronous contraction, electrophysiological coupling, and calcium handling com...
Read More Azerbaijan Medical Journal • 03-10-2024
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